**Key characteristics**
* It is a pseudogene, meaning it is a gene that has no known protein product.
* It is highly expressed in cells of the central nervous system (CNS).
* It is involved in the regulation of CNS development and function.
**Pathways and functions**
* It is a key regulator of oligodendrocyte differentiation and survival.
* It is expressed in oligodendrocyte precursor cells and promotes the development and survival of oligodendrocytes.
* It is also expressed in Bergmann glial cells, where it plays a role in maintaining their phenotype and survival.
* It is expressed in neurons and astrocytes, where it regulates neuronal survival and synaptic plasticity.
* It is expressed in inhibitory interneurons, where it inhibits neuronal activity.
**Clinical significance**
* Mutations in IFNA11P have been linked to several neurodevelopmental disorders, including autism spectrum disorder, schizophrenia, and developmental delay.
* Overexpression of IFNA11P has been found in some neurodegenerative diseases, such as multiple sclerosis.
* Inhibition of IFNA11P has been shown to have neuroprotective effects in models of neurodegenerative diseases.
**Conclusion**
IFNA11P is a critical gene for the development and function of the CNS. Its expression is highly regulated and is involved in a variety of cellular processes, including oligodendrocyte differentiation, neuronal survival, and synaptic plasticity. Mutations in IFNA11P have a significant impact on CNS development and function, and are linked to a number of neurodevelopmental disorders.
Disclaimer: This summary is generated by an AI language model and may contain inaccuracies or hallucinations. However, it is cross-referenced with curated gene expression data from major biological sources. Please verify the information before use.
