Details for: SLC2A1
Associated with
Significant Cells
Cell Significance Index (CSI) scores for the chosen context(s)
-
CSI 30.92rCSI 38.26%PRS 75.54
-
CSI 30.13rCSI 52.11%PRS 68.95
-
CSI 28.09rCSI 43.41%PRS 76.77
-
CSI 21.05rCSI 33.61%PRS 81.39
-
CSI 18.31rCSI 41.78%PRS 69.16
-
CSI 17.51rCSI 34.78%PRS 73.66
-
CSI 17.18rCSI 49.49%PRS 84.43
-
CSI 15.81rCSI 15.25%PRS 70.66
-
CSI 11.88rCSI 18.14%PRS 78.02
-
CSI 11.68rCSI 11.24%PRS 76.99
-
CSI 11.23rCSI 32.13%PRS 85.06
-
CSI 9.18rCSI 45.24%PRS 85.69
-
CSI 8.55rCSI 27.72%PRS 75.65
-
CSI 8.41rCSI 7.39%PRS 66.63
-
CSI 8.05rCSI 9.33%PRS 81.71
-
CSI 7.88rCSI 10.53%PRS 76.53
-
CSI 7.54rCSI 10.45%PRS 75.21
-
CSI 7.26rCSI 6.45%PRS 75.5
-
CSI 7.15rCSI 16.03%PRS 60.29
-
CSI 7.06rCSI 9.66%PRS 82.12
-
CSI 6.8rCSI 5.7%PRS 80.81
-
CSI 5.97rCSI 14.23%PRS 74.78
-
CSI 5.86rCSI 4.34%PRS 71.04
-
CSI 5.84rCSI 31.52%PRS 84.71
-
CSI 5.82rCSI 13.21%PRS 78.6
-
CSI 5.67rCSI 5.91%PRS 77.02
-
CSI 5.52rCSI 7.02%PRS 82.72
-
CSI 5.35rCSI 5.61%PRS 75
-
CSI 5.24rCSI 4.95%PRS 76.58
-
CSI 5.02rCSI 4.37%PRS 87.22
-
CSI 5rCSI 3.49%PRS 81.01
-
CSI 4.88rCSI 5.86%PRS 75.4
-
CSI 4.78rCSI 3.96%PRS 80.18
-
CSI 4.7rCSI 25.7%PRS 74.36
-
CSI 4.58rCSI 6.71%PRS 82.51
-
CSI 4.5rCSI 9.13%PRS 56.12
-
CSI 4.47rCSI 3.71%PRS 78.31
-
CSI 4.38rCSI 7.09%PRS 72.84
-
CSI 4.31rCSI 6.63%PRS 67.96
-
CSI 4.27rCSI 11.33%PRS 84.09
-
CSI 3.88rCSI 3.4%PRS 85.16
-
CSI 3.84rCSI 5.33%PRS 76.44
-
CSI 3.84rCSI 5.62%PRS 72.17
-
CSI 3.77rCSI 3.51%PRS 78.27
-
CSI 3.75rCSI 20.15%PRS 85.77
-
CSI 3.73rCSI 3.66%PRS 80.63
-
CSI 3.7rCSI 2.92%PRS 66.03
-
CSI 3.56rCSI 4.29%PRS 85.46
-
CSI 3.53rCSI 5.79%PRS 67.14
-
CSI 3.37rCSI 2.74%PRS 77.11
-
CSI 3.14rCSI 2.33%PRS 88.36
-
CSI 3.08rCSI 3.55%PRS 70.02
-
CSI 3.05rCSI 3.2%PRS 81.23
-
CSI 2.94rCSI 4.74%PRS 77.27
-
CSI 2.91rCSI 3.73%PRS 81.14
-
CSI 2.83rCSI 2.95%PRS 75.36
-
CSI 2.82rCSI 2.48%PRS 82.78
-
CSI 2.81rCSI 2.8%PRS 71.43
-
CSI 2.79rCSI 4.44%PRS 69.45
-
CSI 2.76rCSI 1.86%PRS 90.06
-
CSI 2.76rCSI 3.36%PRS 84.24
-
CSI 2.73rCSI 5.12%PRS 65.86
-
CSI 2.59rCSI 16.69%PRS 87.8
-
CSI 2.59rCSI 5.04%PRS 80.95
-
CSI 2.54rCSI 2.7%PRS 83.16
-
CSI 2.51rCSI 2.69%PRS 78.78
-
CSI 2.44rCSI 5.57%PRS 71.78
-
CSI 2.3rCSI 2.65%PRS 69.66
-
CSI 2.25rCSI 3.57%PRS 70.53
-
CSI 2.23rCSI 3.05%PRS 77.93
-
CSI 2.22rCSI 3.18%PRS 78.61
-
CSI 2.17rCSI 3.5%PRS 67.62
-
CSI 2.15rCSI 7.27%PRS 76.27
-
CSI 1.92rCSI 3.43%PRS 86.64
-
CSI 1.88rCSI 1.7%PRS 75.9
-
CSI 1.82rCSI 2.78%PRS 74.61
-
CSI 1.82rCSI 4.73%PRS 85.41
-
CSI 1.79rCSI 3.16%PRS 58.75
-
CSI 1.76rCSI 1.82%PRS 82.05
-
CSI 1.74rCSI 2.65%PRS 88.85
-
CSI 1.73rCSI 2.66%PRS 86.15
-
CSI 1.71rCSI 3.26%PRS 91.38
-
CSI 1.66rCSI 2.08%PRS 86.61
-
CSI 1.65rCSI 3.91%PRS 78.55
-
CSI 1.57rCSI 4.08%PRS 79.11
-
CSI 1.51rCSI 3.29%PRS 85.17
-
CSI 1.49rCSI 2.82%PRS 86.02
-
CSI 1.39rCSI 3.53%PRS 67.95
-
CSI 1.39rCSI 3.31%PRS 83.62
-
CSI 1.35rCSI 3.91%PRS 67.07
-
CSI 1.26rCSI 7.79%PRS 87.26
-
CSI 1.21rCSI 3.12%PRS 73.01
-
CSI 1.16rCSI 3.27%PRS 72.76
-
CSI 1.09rCSI 1.45%PRS 83.45
-
CSI 1rCSI 3.79%PRS 68.58
-
CSI 0.95rCSI 5.55%PRS 81.96
-
CSI 0.92rCSI 9.54%PRS 71.7
-
CSI 0.92rCSI 3.66%PRS 86.16
-
CSI 0.9rCSI 3.21%PRS 82.33
-
CSI 0.67rCSI 4.13%PRS 94.24
-
CSI 0.2rCSI 4.8%PRS 89.1%
-
CSI 0.4rCSI 2.2%PRS 89.1%
-
CSI 0.5rCSI 3.0%PRS 83.6%
-
CSI 0.7rCSI 4.1%PRS 94.2%
-
CSI 0.9rCSI 3.2%PRS 82.3%
-
CSI 0.9rCSI 3.7%PRS 86.2%
-
CSI 0.9rCSI 9.5%PRS 71.7%
-
CSI 1.0rCSI 5.6%PRS 82.0%
-
CSI 1.0rCSI 3.8%PRS 68.6%
-
CSI 1.1rCSI 1.5%PRS 83.5%
-
CSI 1.2rCSI 3.3%PRS 72.8%
-
CSI 1.2rCSI 3.1%PRS 73.0%
-
CSI 1.3rCSI 7.8%PRS 87.3%
-
CSI 1.4rCSI 3.9%PRS 67.1%
-
CSI 1.4rCSI 3.3%PRS 83.6%
-
CSI 1.4rCSI 3.5%PRS 68.0%
-
CSI 1.5rCSI 2.8%PRS 86.0%
-
CSI 1.5rCSI 3.3%PRS 85.2%
-
CSI 1.6rCSI 4.1%PRS 79.1%
-
CSI 1.7rCSI 3.9%PRS 78.6%
-
CSI 1.7rCSI 2.1%PRS 86.6%
-
CSI 1.7rCSI 3.3%PRS 91.4%
-
CSI 1.7rCSI 2.7%PRS 86.2%
-
CSI 1.7rCSI 2.7%PRS 88.9%
-
CSI 1.8rCSI 1.8%PRS 82.1%
-
CSI 1.8rCSI 3.2%PRS 58.8%
-
CSI 1.8rCSI 4.7%PRS 85.4%
-
CSI 1.8rCSI 2.8%PRS 74.6%
-
CSI 1.9rCSI 1.7%PRS 75.9%
-
CSI 1.9rCSI 3.4%PRS 86.6%
-
CSI 2.2rCSI 7.3%PRS 76.3%
-
CSI 2.2rCSI 3.5%PRS 67.6%
-
CSI 2.2rCSI 3.2%PRS 78.6%
-
CSI 2.2rCSI 3.1%PRS 77.9%
-
CSI 2.3rCSI 3.6%PRS 70.5%
-
CSI 2.3rCSI 2.7%PRS 69.7%
-
CSI 2.4rCSI 5.6%PRS 71.8%
-
CSI 2.5rCSI 2.7%PRS 78.8%
-
CSI 2.5rCSI 2.7%PRS 83.2%
-
CSI 2.6rCSI 5.0%PRS 81.0%
-
CSI 2.6rCSI 16.7%PRS 87.8%
-
CSI 2.7rCSI 5.1%PRS 65.9%
-
CSI 2.8rCSI 3.4%PRS 84.2%
-
CSI 2.8rCSI 1.9%PRS 90.1%
-
CSI 2.8rCSI 4.4%PRS 69.5%
-
CSI 2.8rCSI 2.8%PRS 71.4%
-
CSI 2.8rCSI 2.5%PRS 82.8%
-
CSI 2.8rCSI 3.0%PRS 75.4%
-
CSI 2.9rCSI 3.7%PRS 81.1%
-
CSI 2.9rCSI 4.7%PRS 77.3%
-
CSI 3.1rCSI 3.2%PRS 81.2%
-
CSI 3.1rCSI 3.6%PRS 70.0%
-
CSI 3.1rCSI 2.3%PRS 88.4%
-
CSI 3.4rCSI 2.7%PRS 77.1%
-
CSI 3.5rCSI 5.8%PRS 67.1%
-
CSI 3.6rCSI 4.3%PRS 85.5%
-
CSI 3.7rCSI 2.9%PRS 66.0%
-
CSI 3.7rCSI 3.7%PRS 80.6%
-
CSI 3.8rCSI 20.2%PRS 85.8%
-
CSI 3.8rCSI 3.5%PRS 78.3%
-
CSI 3.8rCSI 5.6%PRS 72.2%
-
CSI 3.8rCSI 5.3%PRS 76.4%
-
CSI 3.9rCSI 3.4%PRS 85.2%
-
CSI 4.3rCSI 11.3%PRS 84.1%
-
CSI 4.3rCSI 6.6%PRS 68.0%
-
CSI 4.4rCSI 7.1%PRS 72.8%
-
CSI 4.5rCSI 3.7%PRS 78.3%
-
CSI 4.5rCSI 9.1%PRS 56.1%
-
CSI 4.6rCSI 6.7%PRS 82.5%
-
CSI 4.7rCSI 25.7%PRS 74.4%
-
CSI 4.8rCSI 4.0%PRS 80.2%
-
CSI 4.9rCSI 5.9%PRS 75.4%
-
CSI 5.0rCSI 3.5%PRS 81.0%
-
CSI 5.0rCSI 4.4%PRS 87.2%
-
CSI 5.2rCSI 5.0%PRS 76.6%
-
CSI 5.4rCSI 5.6%PRS 75.0%
-
CSI 5.5rCSI 7.0%PRS 82.7%
-
CSI 5.7rCSI 5.9%PRS 77.0%
-
CSI 5.8rCSI 13.2%PRS 78.6%
-
CSI 5.8rCSI 31.5%PRS 84.7%
-
CSI 5.9rCSI 4.3%PRS 71.0%
-
CSI 6.0rCSI 14.2%PRS 74.8%
-
CSI 6.8rCSI 5.7%PRS 80.8%
-
CSI 7.1rCSI 9.7%PRS 82.1%
-
CSI 7.2rCSI 16.0%PRS 60.3%
-
CSI 7.3rCSI 6.5%PRS 75.5%
-
CSI 7.5rCSI 10.5%PRS 75.2%
-
CSI 7.9rCSI 10.5%PRS 76.5%
-
CSI 8.1rCSI 9.3%PRS 81.7%
-
CSI 8.4rCSI 7.4%PRS 66.6%
-
CSI 8.6rCSI 27.7%PRS 75.7%
-
CSI 9.2rCSI 45.2%PRS 85.7%
-
CSI 11.2rCSI 32.1%PRS 85.1%
-
CSI 11.7rCSI 11.2%PRS 77.0%
-
CSI 11.9rCSI 18.1%PRS 78.0%
-
CSI 15.8rCSI 15.3%PRS 70.7%
-
CSI 17.2rCSI 49.5%PRS 84.4%
-
CSI 17.5rCSI 34.8%PRS 73.7%
-
CSI 18.3rCSI 41.8%PRS 69.2%
-
CSI 21.1rCSI 33.6%PRS 81.4%
Fold Change: Represents the ratio of the current Cell Significance Index to the Cell Significance Index Threshold, indicating how much the gene expression has changed compared to a baseline.
Cell Significance Index: Reflects how strongly a gene is expressed in this specific cell.
Fold Change: Represents the ratio of the current Cell Significance Index to the Cell Significance Index Threshold, indicating how much the gene expression has changed compared to a baseline.
Cell Significance Index: Reflects how strongly a gene is expressed in this cell type. Calculated using techniques like effect size estimation and bootstrapping for reliability.
Fold Change: Represents the ratio of the current Cell Significance Index to the Cell Significance Index Threshold, indicating how much the gene expression has changed compared to a baseline.
Cell Significance Index: Reflects how strongly a gene is expressed in this cell type. Calculated using techniques like effect size estimation and bootstrapping for reliability.
Network Configuration
Explore relationships of the current gene. Select an Interaction Source: 'ONTOLOGY' for shared pathways (GO/Reactome) or 'STRING' for protein-protein interactions. Further refine by selecting context genes and comparing Cell Significance Index (CSI) scores between baseline and target cell types and their specific contexts.
Legend:
- Query Gene
-
Node Color (Target Cell CSI, relative to current network):
- Very High
- High
- Medium
- Low
- Very Low
- CSI N/A
- Node Size: Proportional to Target Cell CSI magnitude
- STRING PPI Edge
- Shared Pathway Edge (ONTOLOGY)
Other Information
This section provides additional information about the gene, including a description generated by an AI language model and details about associated proteins.
Genular Protein ID: 4196089677
Symbol: GTR1_HUMAN
Name: N/A
UniProtKB Accession Codes:
Database IDs:
Citations:
PubMed ID: 3839598
Title: Sequence and structure of a human glucose transporter.
PubMed ID: 3839598
PubMed ID: 14702039
Title: Complete sequencing and characterization of 21,243 full-length human cDNAs.
PubMed ID: 14702039
DOI: 10.1038/ng1285
PubMed ID: 15489334
Title: The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC).
PubMed ID: 15489334
DOI: 10.1101/gr.2596504
PubMed ID: 2834252
Title: Characterization and expression of human HepG2/erythrocyte glucose-transporter gene.
PubMed ID: 2834252
PubMed ID: 10954735
Title: Hexose permeation pathways in Plasmodium falciparum-infected erythrocytes.
PubMed ID: 10954735
PubMed ID: 17081065
Title: Proteomic and bioinformatic characterization of the biogenesis and function of melanosomes.
PubMed ID: 17081065
DOI: 10.1021/pr060363j
PubMed ID: 18245775
Title: Transmembrane segment 6 of the Glut1 glucose transporter is an outer helix and contains amino acid side chains essential for transport activity.
PubMed ID: 18245775
PubMed ID: 18347014
Title: Dematin and adducin provide a novel link between the spectrin cytoskeleton and human erythrocyte membrane by directly interacting with glucose transporter-1.
PubMed ID: 18347014
PubMed ID: 18691976
Title: Kinase-selective enrichment enables quantitative phosphoproteomics of the kinome across the cell cycle.
PubMed ID: 18691976
PubMed ID: 19449892
Title: Model of the exofacial substrate-binding site and helical folding of the human Glut1 glucose transporter based on scanning mutagenesis.
PubMed ID: 19449892
DOI: 10.1021/bi900521n
PubMed ID: 19349973
Title: Mass-spectrometric identification and relative quantification of N-linked cell surface glycoproteins.
PubMed ID: 19349973
DOI: 10.1038/nbt.1532
PubMed ID: 20068231
Title: Quantitative phosphoproteomics reveals widespread full phosphorylation site occupancy during mitosis.
PubMed ID: 20068231
PubMed ID: 21269460
Title: Initial characterization of the human central proteome.
PubMed ID: 21269460
PubMed ID: 21406692
Title: System-wide temporal characterization of the proteome and phosphoproteome of human embryonic stem cell differentiation.
PubMed ID: 21406692
PubMed ID: 22814378
Title: N-terminal acetylome analyses and functional insights of the N-terminal acetyltransferase NatB.
PubMed ID: 22814378
PubMed ID: 23219802
Title: Stomatin interacts with GLUT1/SLC2A1, band 3/SLC4A1, and aquaporin-1 in human erythrocyte membrane domains.
PubMed ID: 23219802
PubMed ID: 23186163
Title: Toward a comprehensive characterization of a human cancer cell phosphoproteome.
PubMed ID: 23186163
DOI: 10.1021/pr300630k
PubMed ID: 23563491
Title: A global analysis of SNX27-retromer assembly and cargo specificity reveals a function in glucose and metal ion transport.
PubMed ID: 23563491
DOI: 10.1038/ncb2721
PubMed ID: 24275569
Title: An enzyme assisted RP-RPLC approach for in-depth analysis of human liver phosphoproteome.
PubMed ID: 24275569
PubMed ID: 25957687
Title: Rod-derived cone viability factor promotes cone survival by stimulating aerobic glycolysis.
PubMed ID: 25957687
PubMed ID: 25982116
Title: A protein kinase C phosphorylation motif in GLUT1 affects glucose transport and is mutated in GLUT1 deficiency syndrome.
PubMed ID: 25982116
PubMed ID: 32860739
Title: Structural Basis for Blocking Sugar Uptake into the Malaria Parasite Plasmodium falciparum.
PubMed ID: 32860739
PubMed ID: 35810171
Title: A Nodal enhanced micropeptide NEMEP regulates glucose uptake during mesendoderm differentiation of embryonic stem cells.
PubMed ID: 35810171
PubMed ID: 24847886
Title: Crystal structure of the human glucose transporter GLUT1.
PubMed ID: 24847886
DOI: 10.1038/nature13306
PubMed ID: 27078104
Title: Mechanism of inhibition of human glucose transporter GLUT1 is conserved between cytochalasin B and phenylalanine amides.
PubMed ID: 27078104
PubMed ID: 10227690
Title: Defective glucose transport across brain tissue barriers: a newly recognized neurological syndrome.
PubMed ID: 10227690
PubMed ID: 10980529
Title: Mutational analysis of GLUT1 (SLC2A1) in Glut-1 deficiency syndrome.
PubMed ID: 10980529
DOI: 10.1002/1098-1004(200009)16:3<224::aid-humu5>3.0.co;2-p
PubMed ID: 11603379
Title: Autosomal dominant Glut-1 deficiency syndrome and familial epilepsy.
PubMed ID: 11603379
DOI: 10.1002/ana.1222
PubMed ID: 11136715
Title: Autosomal dominant transmission of GLUT1 deficiency.
PubMed ID: 11136715
DOI: 10.1093/hmg/10.1.63
PubMed ID: 12325075
Title: Imaging the metabolic footprint of Glut1 deficiency on the brain.
PubMed ID: 12325075
DOI: 10.1002/ana.10311
PubMed ID: 14605501
Title: GLUT-1 deficiency without epilepsy -- an exceptional case.
PubMed ID: 14605501
PubMed ID: 15622525
Title: Glut-1 deficiency syndrome: clinical, genetic, and therapeutic aspects.
PubMed ID: 15622525
DOI: 10.1002/ana.20331
PubMed ID: 18451999
Title: GLUT1 mutations are a cause of paroxysmal exertion-induced dyskinesias and induce hemolytic anemia by a cation leak.
PubMed ID: 18451999
DOI: 10.1172/jci34438
PubMed ID: 19798636
Title: Early-onset absence epilepsy caused by mutations in the glucose transporter GLUT1.
PubMed ID: 19798636
DOI: 10.1002/ana.21724
PubMed ID: 19901175
Title: Childhood chorea with cerebral hypotrophy: a treatable GLUT1 energy failure syndrome.
PubMed ID: 19901175
PubMed ID: 19630075
Title: GLUT1 gene mutations cause sporadic paroxysmal exercise-induced dyskinesias.
PubMed ID: 19630075
DOI: 10.1002/mds.22507
PubMed ID: 20221955
Title: Autosomal recessive inheritance of GLUT1 deficiency syndrome.
PubMed ID: 20221955
PubMed ID: 20129935
Title: Glucose transporter-1 deficiency syndrome: the expanding clinical and genetic spectrum of a treatable disorder.
PubMed ID: 20129935
DOI: 10.1093/brain/awp336
PubMed ID: 21204808
Title: Mild adolescent/adult onset epilepsy and paroxysmal exercise-induced dyskinesia due to GLUT1 deficiency.
PubMed ID: 21204808
PubMed ID: 20621801
Title: Paroxysmal exercise-induced dyskinesia, writer's cramp, migraine with aura and absence epilepsy in twin brothers with a novel SLC2A1 missense mutation.
PubMed ID: 20621801
PubMed ID: 20574033
Title: Absence epilepsies with widely variable onset are a key feature of familial GLUT1 deficiency.
PubMed ID: 20574033
PubMed ID: 21791420
Title: Stomatin-deficient cryohydrocytosis results from mutations in SLC2A1: a novel form of GLUT1 deficiency syndrome.
PubMed ID: 21791420
PubMed ID: 20830593
Title: Excellent response to acetazolamide in a case of paroxysmal dyskinesias due to GLUT1-deficiency.
PubMed ID: 20830593
PubMed ID: 21832227
Title: Paroxysmal choreoathetosis/spasticity (DYT9) is caused by a GLUT1 defect.
PubMed ID: 21832227
PubMed ID: 23280796
Title: Glucose transporter 1 deficiency in the idiopathic generalized epilepsies.
PubMed ID: 23280796
DOI: 10.1002/ana.23702
PubMed ID: 22492876
Title: An infant with pseudohyperkalemia, hemolysis, and seizures: cation-leaky GLUT1-deficiency syndrome due to a SLC2A1 mutation.
PubMed ID: 22492876
DOI: 10.1210/jc.2012-1399
PubMed ID: 22282645
Title: GLUT1 mutations are a rare cause of familial idiopathic generalized epilepsy.
PubMed ID: 22282645
PubMed ID: 30197081
Title: Mutations in disordered regions can cause disease by creating dileucine motifs.
PubMed ID: 30197081
Sequence Information:
- Length: 492
- Mass: 54084
- Checksum: E71E1C6BD1B00B1E
- Sequence:
MEPSSKKLTG RLMLAVGGAV LGSLQFGYNT GVINAPQKVI EEFYNQTWVH RYGESILPTT LTTLWSLSVA IFSVGGMIGS FSVGLFVNRF GRRNSMLMMN LLAFVSAVLM GFSKLGKSFE MLILGRFIIG VYCGLTTGFV PMYVGEVSPT ALRGALGTLH QLGIVVGILI AQVFGLDSIM GNKDLWPLLL SIIFIPALLQ CIVLPFCPES PRFLLINRNE ENRAKSVLKK LRGTADVTHD LQEMKEESRQ MMREKKVTIL ELFRSPAYRQ PILIAVVLQL SQQLSGINAV FYYSTSIFEK AGVQQPVYAT IGSGIVNTAF TVVSLFVVER AGRRTLHLIG LAGMAGCAIL MTIALALLEQ LPWMSYLSIV AIFGFVAFFE VGPGPIPWFI VAELFSQGPR PAAIAVAGFS NWTSNFIVGM CFQYVEQLCG PYVFIIFTVL LVLFFIFTYF KVPETKGRTF DEIASGFRQG GASQSDKTPE ELFHPLGADS QV
Genular Protein ID: 3480551254
Symbol: Q59GX2_HUMAN
Name: N/A
UniProtKB Accession Codes:
Database IDs:
Sequence Information:
- Length: 517
- Mass: 57018
- Checksum: 0A15C476A80ABF02
- Sequence:
AGERRSHARR HPRTRRSQSH QRSAAMEPSS KKLTGRLMLA VGGAVLGSLQ FGYNTGVINA PQKVIEEFYN QTWVHRYGES ILPTTLTTLW SLSVAIFSVG GMIGSFSVGL FVNRFGRRNS MLMMNLLAFV SAVLMGFSKL GKSFEMLILG RFIIGVYCGL TTGFVPMYVG EVSPTALRGA LGTLHQLGIV VGILIAQVFG LDSIMGNKDL WPLLLSIIFI PALLQCIVLP FCPESPRFLL INRNEENRAK SVLKKLRGTA DVTHDLQEMK EESRQMMREK KVTILELFRS PAYRQPILIA VVLQLSQQLS GINAVFYYST SIFEKAGVQQ PVYATIGSGI VNTAFTVVSL FVVERAGRRT LHLIGLAGMA GCAILMTIAL ALLEQLPWMS YLSIVAIFGF VAFFEVGPGP IPWFIVAELF SQGPRPAAIA VAGFSNWTSN FIVGMCFQYV EQLCGPYVFI IFTVLLVLFF IFTYFKVPET KGRTFDEIAS GFRQGGASQS DKTPEELFHP LGADSQV